Athletic performance
Variants associated with muscle fibre type, response to training and recovery. It is the part of the report with the most uneven evidence, and that is why each card carries its level.

All 12 of your performance traits, with their support stated
Strength ↔ endurance profile
The position combines the variants that contribute to this axis. The scale is only indicative: the differences between genotypes are small and overlap a great deal.
Your summary: 12 traits read — 1 with solid support and 11 with weaker support. Each card states its level of evidence.
Early muscle fatigue
C/CThe variant in homozygosity causes AMP deaminase deficiency, which can cause fatigue and cramps with intense exercise. You do not carry it.
Muscle fiber type
R/XIt is the most studied variant in sports genetics. The RR genotype is common among elite sprinters and XX among distance runners, but the overlap is enormous: there are Olympic champions with all three genotypes. With R/X the profile is intermediate and does not point toward any particular sport.
Aerobic response to training
G/AIt is involved in the production of mitochondria in muscle, which is what improves with endurance training. The measured effect is small compared with how much and how you train.
Risk of tendon and ligament injury
C/TIt affects the structure of type V collagen, present in tendons and ligaments. It has been associated with anterior cruciate ligament injury and with Achilles tendinopathy. It is one of the few items in this section with a concrete action behind it: eccentric work and careful load progression, which are worth doing with any genotype anyway.
Caffeine as an ergogenic aid
A/CIn poor metabolizers, caffeine before competing works less well —and in some studies even worsens performance— compared with rapid metabolizers, where it does work. This is consistent with the result in the nutrigenetics section.
Endurance component (ACE)
No evaluableThe ACE I/D polymorphism is a 287 base pair insertion, not a single-letter change. A genotyping array does not see it: it requires a specific PCR. It is reported anyway because it usually appears in commercial sports panels.
Ligament and tendon strength
G/AThe COL1A1 promoter variant has been associated with a lower risk of cruciate ligament rupture and shoulder dislocation. It is one of the few associations in this block that has replicated in independent cohorts.
Recovery and inflammation
G/CIt modulates the inflammatory response after exercise, which is part of the adaptation process. The evidence on recovery times is preliminary.
Cardiovascular response to exercise
G/AIt has been associated with differences in the vascular response to exercise and in heart rate recovery. The studies are small and do not always agree.
Vasodilation during exertion
T/CIt has been associated with lower nitric oxide production and, in some papers, with lower endurance performance. The evidence is limited.
Adaptation to altitude and to low oxygen
C/TIt has been described as more frequent in power athletes and has been associated with a different response to altitude training. These are small studies in specific populations.
Resting energy expenditure
A/GIt has been associated with slightly lower resting energy expenditure and with somewhat more difficulty losing weight in some studies. The measured effect is small and replication has been uneven.
No study has shown that knowing these genotypes improves an athletic result, or that it helps in choosing a sport or designing a training plan. The individual effects are small and are buried under training, rest, nutrition, and technique. It is reported as material of interest, not as a training guide — and anyone selling you 'your ideal sport according to your DNA' is selling you something the evidence does not support.